MEIS2 Is an Oncogenic Partner in AML1-ETO-Positive AML.

نویسندگان

  • Naidu M Vegi
  • Josef Klappacher
  • Franz Oswald
  • Medhanie A Mulaw
  • Amit Mandoli
  • Verena N Thiel
  • Shiva Bamezai
  • Kristin Feder
  • Joost H A Martens
  • Vijay P S Rawat
  • Tamoghna Mandal
  • Leticia Quintanilla-Martinez
  • Karsten Spiekermann
  • Wolfgang Hiddemann
  • Konstanze Döhner
  • Hartmut Döhner
  • Hendrik G Stunnenberg
  • Michaela Feuring-Buske
  • Christian Buske
چکیده

Homeobox genes are known to be key factors in leukemogenesis. Although the TALE family homeodomain factor Meis1 has been linked to malignancy, a role for MEIS2 is less clear. Here, we demonstrate that MEIS2 is expressed at high levels in patients with AML1-ETO-positive acute myeloid leukemia and that growth of AML1-ETO-positive leukemia depends on MEIS2 expression. In mice, MEIS2 collaborates with AML1-ETO to induce acute myeloid leukemia. MEIS2 binds strongly to the Runt domain of AML1-ETO, indicating a direct interaction between these transcription factors. High expression of MEIS2 impairs repressive DNA binding of AML1-ETO, inducing increased expression of genes such as the druggable proto-oncogene YES1. Collectively, these data describe a pivotal role for MEIS2 in AML1-ETO-induced leukemia.

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عنوان ژورنال:
  • Cell reports

دوره 16 2  شماره 

صفحات  -

تاریخ انتشار 2016